Letter to the editor
Recurrent Takotsubo cardiomyopathy presenting with different morphologic patterns

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Acknowledgement

The authors of this manuscript have certified that they comply with the Principles of Ethical Publishing in the International Journal of Cardiology [6].

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Cited by (16)

  • Change of heart: Reverse takotsubo's cardiomyopathy – A case report

    2021, Clinical Imaging
    Citation Excerpt :

    The exact mechanism of development of TTC is unknown, some of the proposed theories include catecholamine induced, coronary artery spasm, coronary microvascular impairment, and estrogen deficiency. Differences in the number and location of adrenoreceptor concentrations have been hypothesized as a cause for the difference in distribution of regional wall abnormality between the variants and classic TTC [7,8]. Patients with rTTC present at an earlier age, due to the abundance of adrenoceptors at the base compared to the apex [5].

  • Takotsubo syndrome

    2018, Indian Heart Journal
    Citation Excerpt :

    CA 125 could thus be a useful marker for early risk stratification in patients with TS. Anatomical variations in sympathetic innervations and adrenergic receptor density with differential dynamic response to emotional/physical stress has been suggested as another hypothesis for Takotsubo syndrome.31,32 High local concentration of nor-epinephrine might results in apical hypokinesia and increasing mechanical stress at the apex.

  • Updates on publication trends in Takotsubo syndrome

    2016, International Journal of Cardiology
  • Differences between Takotsubo cardiomyopathy and reverse Takotsubo cardiomyopathy associated with subarachnoid hemorrhage

    2016, IJC Heart and Vasculature
    Citation Excerpt :

    Several authors have attempted to identify potential differences between TTC and r-TTC unrelated to neurological diseases, and reported several differences [3,4]. Meanwhile, other authors have argued that TTC and r-TTC may be a temporal evolution of the same disease, based on patients who developed r-TTC several weeks to months after an episode of typical TTC [5–8]. Therefore, the question of whether TTC is different from r-TTC has not been answered fully.

  • Four episodes of takotsubo cardiomyopathy in one patient

    2016, International Journal of Cardiology
  • Takotsubo cardiomyopathy triggered by lateral wall ST-segment elevation myocardial infarction

    2014, Journal of Cardiology Cases
    Citation Excerpt :

    Interestingly, in the present case, the first TC involved apical ballooning, while the second TC was characterized by mid-ventricular ballooning. Recurrent TC with different patterns of left ventricular involvement has been previously reported [13–15], which raises a question regarding the currently proposed hypothesis of variability in beta-adrenergic receptor density and sensitivity in the pathogenesis of TC [15]. This is the second case report of TC triggered by an acute MI.

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